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Calton (Citalopram)

Calton is used to treat depression. Calton is in a class of antidepressants called selective serotonin reuptake inhibitors (SSRIs). It is thought to work by increasing the amount of serotonin, a natural substance in the brain that helps maintain mental balance. Calton comes as a tablet and a solution (liquid) to take by mouth. It is usually taken once a day, in the morning or in the evening, with or without food. Take Calton at around the same time every day. Follow the directions on your prescription label carefully, and ask your doctor or pharmacist to explain any part you do not understand. Take Calton exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor.

Other names for this medication:
Citalopram, Celexa, Akarin, Arpolax, Aurex, Belmazol, Calton, Celapram, Celius, Ciazil, Cilift, Cimal, Ciprager, Cipram, Cipramil, Ciprapine, Ciprotan, Cital, Citalec, Citapram, Citara, Citaxin, Citol, Citopam, Citox, Citrex, Citrol, Citronil, Dalsan, Depramil, Elopram, Eostar, Humorap, Hydertan, Laira, Lopram, Lopraxer, Opra, Oropram, Prisdal, Proximax, Relaxol, Sepram, Seropram, Somac, Talohexal, Talosin, Temperax, Verisan, Vodelax, Zebrak, Zentius,

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Sanprima, Practon, Zoldem, Betapace, Rebetron, Arcamox, Contracid, Metrogyl, Amlozek, Zenil, Feloran, Clarimax, Alpram, Imped, Amosine, Alprax, Fenactol, Trandate, Disipan, Exten, Diclomol, Efexor, Artren, Klomeprax, Lipidem, Dolofarmalan, Ramipril, Coversum, Tervalon, Peptidin, Flogene, Antadine, Finasept,

Calton is a type of antidepressant known as a selective serotonin reuptake inhibitor (SSRI).

It's often used to treat depression and also sometimes for panic attacks.

Calton helps many people recover from depression, and has fewer unwanted side effects than older antidepressants.

Calton is available on prescription as tablets and liquid drops that you put in a drink of water.

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Virus-host coevolution: common patterns of nucleotide motif usage in Flaviviridae and their hosts. In contrast to humans, where non-myeloid cells utilize both promoters, the distal Tlr4 promoter initiates all Tlr4 transcripts in murine non-myeloid cells, indicating that species-specific differences in TLR4 mRNA regulation may primarily exist in non-myeloid cell types. Most of the research in clinical trials is based on longitudinal designs, which involve repeated measurements of a variable of interest. These findings provides the basis for developing strategies to increase systemic levels of free ISG15 to treat cancer patients overexpressing the ISG15 pathway.

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