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Saitan (Telmisartan)
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Telmisartan

At 24 weeks of age, the treatment with Saitan dose-dependently ameliorated hyperglycemia and hypoinsulinemia, and high-dose (5 mg/kg/day) treated SDT rats did not developed diabetes. The effects of calcium antagonists (amlodipine) and angiotensin II receptor antagonists (Saitan) on lipid profile and oxidative markers were investigated in Algerian hypertensive patients.

Other names for this medication:
Telmisartan, Micardis, Kinzalkomb, Kinzalmono, Mitosan, Pritor, Pritorplus, Saitan, Telma, Telpres, Telsan, Twynsta,

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Saitan comes as a tablet to take by mouth. It is usually taken once a day with or without food. To help you remember to take Saitan, take it around the same time every day. Follow the directions on your prescription label carefully, and ask your doctor or pharmacist to explain any part you do not understand. Take Saitan exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor.

Saitan tablets come in individual blister packs that can be opened by peeling back the paper layer from the foil and pushing the tablet through the foil. Do not open a blister pack until you are ready to swallow the tablet it contains.

Your doctor may start you on a low dose of Saitan and gradually increase your dose.

Saitan controls high blood pressure but does not cure it. Your blood pressure may decrease during the first 2 weeks of your treatment, but it may take 4 weeks for you to notice the full benefit of Saitan. Continue to take Saitan even if you feel well. Do not stop taking Saitan without talking to your doctor.

Ask your pharmacist or doctor for a copy of the manufacturer's information for the patient.

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Changes in hospital readmissions for diabetes-related conditions. DAPC and genetic distance estimation indicated that the dIBDVs are one of the most genetically divergent IBDV lineages. The clinical symptoms and serologic status of 17 patients with specific IgE and positive skin prick test results to A.s. Characterization of the excitatory mechanism induced by Jingzhaotoxin-I inhibiting sodium channel inactivation. Measurements were compared to readings of two digital manometers simultaneously measuring real IOP in the anterior chamber and vitreous cavity.

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Eosinophilic Coronary Periarteritis with Arterial Dissection: The Mast Cell Hypothesis. Iridium-catalyzed reductive carbon-carbon bond cleavage reaction on a curved pyridylcorannulene skeleton. There is no consensus regarding the optimal treatment for patients with acute Achilles tendon rupture.

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